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肿瘤与下丘脑间有胶质增生层。A gliosis layer between the wall of tumor and the hypothalamus was seen.
这些图片仅仅是反应性神经胶质增生，没有肿瘤或特别的异常之处。These photos show only reactive gliosis and no neoplasm or specific abnormalities.
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MRI可以显示早期缺血及胶质增生的征像，但随时间延长脑实质出现萎缩。MRI may show signs of ischemia and gliosis in early stages, but parenchymal atrophy over time.
回顾性分析经手术与病理证实的13例胶质细胞增生症病人的临床资料。Methods Clinical data of 13 patients with pathologically confirmed gliosis were analyzed retrospectively.
病变的信号特点很可能是因为错构瘤区域致密的胶质增生和髓鞘都是造成的。The signal characteristics probably result from dense gliosis and diminished myelin sheaths in the hemartomatous areas.
目的构建反义及正义波形蛋白重组逆转录病毒载体，研究波形蛋白在反应性胶质化中的功能与作用。Objective To construct antisense and sense vimentin retrovirus vector for investigating the effect of vimentin on reactive gliosis.
这种神经元的丢失通常伴有星形胶质细胞增生、神经胶质过多症以及特定的神经病理标记，如帕金森病的路易小体。This neuronal loss is frequently accompanied with astrocytosis, gliosis and specific neuropathologic markers, such as Lewy body in Parkinson's disease.
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波形蛋白是主要存在于间充质细胞中的一种中间丝，与脑损伤后的反应性胶质化和瘢痕修复密切相关。Vimentin, one type of intermediate filaments, mainly exists in mesenchymal cells and is closely related to gliosis and glial scar recovery after brain injury.
脑穿通囊肿通常是实质内并且与脑室相交通，周边脑实质萎缩并伴有胶质增生。Porencephalic cysts are usually intraparenchymal and communicate with the ventricle. The surrounding brain parenchyma will be atrophic with accompanying gliosis.
结果难治性颞叶癫痫患者颞叶组织胶质细胞呈不同程度增生，部分有噬神经元现象和神经元变性。Results Gliosis was observed, to different extent, in the brain tissues from patients with refractory temporal lobe epilepsy, so were neuronophagia phenomenon and neuronal degeneration.