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甲状腺激素替代治疗后可改善低氧和高碳酸血症。The hypoxia and hypercapnia ameliorated after thyroid hormone therapy.

股静脉注射纳洛酮使高二氧化碳通气反应增强。Ventilation response to hypercapnia was enhanced by injection of naloxone into femoral vein.

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结论慢性低氧高二氧化碳诱导了大鼠支气管血栓素合酶基因的表达。Conclusion The expression of TXS gene in bronchi was induced by chronic hypoxia and hypercapnia.

结论慢性低氧高二氧化碳诱导了大鼠支气管前列环素合酶基因的表达。Conclusions Expression of PGI_2 S in bronchi rats should be induced by chronic hypoxia and hypercapnia.

结论腹腔海水浸泡可引起机体出现低氧血症、高碳酸血症及酸中毒。Conclusions Seawater immersion after open abdominal wound can result in anoxemia, hypercapnia and acidosis.

低氧和高碳酸血证时增加通气量是临床上有用的信号也是内环境稳态的反应。An increase in ventilation during hypoxia and hypercapnia is a useful clinical sign and also a homeostatic response.

这些情况可能是由于疼痛所致，尽管相似的体症也可能与麻醉性镇痛药引起通气不足和高碳酸血症有关。These findings may be due to pain, although similar signs are associated with inadequate ventilation and hypercapnia compounded by the use of narcotics.

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结论对中间型综合征患者，停机后如果有单纯轻、中度二氧化碳滞留，可不必继续作机械通气。Conclusion Continuous mechanical ventilation was not required for the IMS patients with simple light and moderate hypercapnia when weaning from mechanical ventilation.

股静脉注射羟甲芬太尼对低氧通气反应无影响，对高二氧化碳通气反应有明显的抑制作用。Injection of ohmefentanyl into femoral vein had no effect on hypoxia induced ventilatory response, but inhibited the ventilatory response to hypercapnia significantly.

结果低氧高碳酸血症条件下，心交感神经传出冲动发放频率明显增加，心肌耗氧量增加，心肌收缩能力增强。Results The simulation experiments show that cardiac sympathetic activities, cardiac oxygen consumption and myocardiac contractility increased during hypercapnia hypoxia.

叙述了实施允许性高碳酸血症的可行性和碳酸血症的高限，强调高碳酸血症并非通气追求的最终目标。It also describes feasibility of permissive hypercapnia ventilation and top limit of carbonemia . It emphasises that hypercapnia is not the final goal seeked by ventilation.

呼吸道感染是肺心病急性发作的诱因，而由此引起和加重低氧血症和高碳酸血症是导致多脏器损伤的主要原因。The attack of pulmonary heart disease was caused by respiratory tract infection. And that resulted in and exacerbated the hypoxemia and hypercapnia which caused the injury of organs.

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起初动脉的氧合可能是正常的，也可能出现轻微的低碳酸血症，随着阻塞的加重，病人则出现典型的低氧血症和高碳酸血症。Whereas initially arterial oxygenation may be normal and slight hypocapnia may be present, worsening obstruction and tiring patients may exemplify arterial hypoxemia and hypercapnia.