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结论镓盐对骨质疏松引起的骨细胞超微结构的损伤有改善作用。Gallium can alleviate the damage of osteocyte ultrastructure elicited by osteoporosis.

目的探讨镓盐对骨质疏松骨细胞超微结构的影响。Objective to investigate the influence of gallium on osteocyte ultrastructure in osteoporosis.

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电镜发现骨小梁内早期即出现幼稚骨细胞和胶原原纤维合成。Also juvenal osteocyte and collagen fibril were observed in early stage by electron microscope.

目的探讨镓盐对骨质疏松骨细胞超微结构的影响。Conclusion Gallium can alleviate the damage of osteocyte ultrastructure elicited by osteoporosis.

结论镓盐对骨质疏松引起的骨细胞超微结构的损伤有改善作用。Conclusion gallium can alleviate the damage of osteocyte ultrastructure elicited by osteoporosis.

骨细胞形态和骨结构间的确切关系还很复杂,值得进一步加以研究。The exact relationship between osteocyte morphology and bone architecture, however, is complex and deserves further study.

本研究探讨骨细胞形态在不同BMD条件下的不同骨病理中可能起到的作用。In this study, we explored the possibility that osteocyte morphology might play a role in various bone pathologies with different BMD.

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为更好认识骨细胞在骨组织中的作用,作者对骨细胞在骨组织中的网络结构进行了综述。This review is intended to synthesize current understanding of osteocyte network that will promote understanding of this obscure cell.

为帮助骨细胞存活,医师在获取自体松质骨时应该将骨细胞保存在潮湿低温的浸血海绵中。In order to improve osteocyte survival when obtaining autogenous cancellous bone, the surgeon should keep the donor cells moist and chilled in a blood- soaked sponge.

糖皮质激素性骨质疏松症的发病机制可能是其损害肠道的钙吸收,严重抑制成骨形成,刺激骨细胞的凋亡。Recently it is suggested that glucocorticoid therapy might impair intestinal calcium absorption, dramatically suppress osteoblastic formation, and stimulate osteocyte apoptosis.

生物活性玻璃具有良好的骨传导性、诱导性,能加速骨细胞的增殖,使成骨细胞转变为成熟的骨细胞。The biological activity glass has the good bone conduction, the inductivity, can accelerate osteocyte multiplication, causes the osteoblasts to transform into the mature osteocytes.

它具有多向分化潜能的特性,在特定的条件下,可以分化为骨细胞、软骨细胞、肌腱细胞和脂肪细胞等。It has potential of multi-directional differentiation, and can differentiated into many cells in specific condition, such as osteocyte , chondrocyte, tendon cell, lipocyte and so on.