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血管周围的淋巴细胞主要是T辅助。Perivascular T lymphocytes are mainly auxiliary.

因为胶质瘤不会有血管周围套特征。Since gliomas donot give this perivascular cuff feature.

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肿瘤细胞的生长就像围绕血管的袖套。Tumor cells grow as a perivascular cuff around a blood vessel.

你们看到血管周炎症或嗜酸颗粒小体了吗？Did you see perivascular inflammation or eosinophilic granular bodies?

PML有髓鞘丢失、相关的巨噬细胞以及血管周围炎症细胞。PML lesions have myelin loss, associated macrophages, even the perivascular inflammation.

实质细胞间存在与血管周隙相通的细胞间小管。Between parenchymal cells existed an intercellular canaliculus linking perivascular space.

而后者主要是因为外膜慢性炎症以及血管周围过多的胶原沉着引起。The latter is due to adventitial chronic inflammation and excessive perivascular collagen deposition.

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当VEGF活性受到损坏，肿瘤细胞侵入大脑血管管腔的周围。When VEGF activity is impaired, tumor cells invade deep into the brain in the perivascular compartment.

结论EAE的病理改变主要为血管周围炎性浸润及白质脱髓鞘。Conclusion EAE is characterised by perivascular inflammatory cell infiltration and demyelination in white matter.

血管周围间隙扩大通常发生于基底节区、中脑、丘脑和深部脑白质。Dilated perivascular spaces most commonly occur within the basal ganglia, midbrain, thalami, and deep white matter.

因为此为血管周围间隙扩大的典型表现，所以无需进一步影像学检查。Since the appearance of these findings is typical for dilated perivascular spaces, no further imaging is necessary.

这些血管变化干扰了营养物质从血浆中向血管周围邻接的组织和细胞扩散。The vascular changes interfere with diffusion of nutrients from plasma into adjacent perivascular tissue and cells.

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游出是指白细胞从血管内进入血管周围组织的过程。Emigration refers to the process by which motile white cells escape from the blood vessels to the perivascular tissues.

风团活检显示真皮下部组织水肿和血管周围有细胞浸润，主要为单一核细胞、嗜酸性粒细胞。Wind Mission lower biopsy showed dermal edema and infiltration of perivascular cells, mainly mononuclear cells, eosinophils.

结论套环能加速载脂蛋白E基因敲除小鼠颈总动脉斑块形成。Conclusions Perivascular common carotid collar placement can accelerate atherosclerotic plaque formation in apolipoprotein E-knock out mice.

血管周围炎症细胞浸润主要是小淋巴细胞而未见坏死或者其他中枢神经性炎症时常见的其他征象。The perivascular inflammatory infiltrates are mainly small lymphocytes without associated necrosis or other features of usual CNS lymphomas.

结果提示，PGE2与BK可能是引起血管周围感觉神经兴奋和CGRP释放的主要炎症介质。Our data indicate that PGE2 and BK are the major inflammatory mediators in triggering the release of CGRP from the perivascular CGRP-containing nerve.

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本研究我们观察了在载脂蛋白E缺失小鼠中，血管周围肥大细胞对于动脉粥样硬化形成以及斑块稳定性的影响。In the present study, we investigated the contribution of perivascular mast cells to atherogenesis and plaque stability in apolipoprotein E–deficient mice.

血管增生扩张、血管周围淋巴细胞浸润、出血、坏死、囊性变常见。Vascular proliferation, angiectasis, perivascular lymphocyte infiltration, hemorrhage, necrosis, cystoid degeneration and calcification were seen frequently.

实质细胞显示了丰富的线粒体和明显的滑面内质网，也具有粗面内质网、核糖体、高尔基氏器、微管和微丝等。The parenchymal cells have numerous processes, whose terminals are usually found within the perivascular space and contain both clear and dense-core vesicles.