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这一结果为开展口腔癌的HPV病因学研究提供了参考资料。The results provided data for the study on HPV virological oncogenesis of the oral Cancers.

结论端粒缩短并激活端粒酶是涎腺肿瘤形成的重要因素。Conclusion Telomere reduction and telomerase activation are critical factor during oncogenesis.

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由于错配修复基因缺陷而导致的基因组不稳定，被认为是导致肿瘤发生的一条新途径。The instability of genome which results from defect of MMR genes is thought as a new oncogenesis way.

PML是一个生长和转录抑制因子，研究表明PML表达变化可能和一些实体癌的发生有关。PML is a growth and transformation suppressor, and it may play a role in the oncogenesis of solid tumors.

尽管有很多HPV血清型，其中两种—16和18—在引发肿瘤中占绝大多数。Although there are many HPV serotypes, two of them — 16 and 18 — account for the lion's share of the oncogenesis.

充实的证据表明炎症能够促进肿瘤发生，偶尔也会参与癌症排斥。Substantial evidence shows that inflammation promotes oncogenesis and, occasionally, participates in cancer rejection.

端粒酶激活是细胞永生化和肿瘤形成的关键步骤，其活性受到多种因素的调控。Activation of telomerase is the critical step in cellular immortality and oncogenesis. Its activity is controled by many factors.

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环氧合酶是一种完整的膜结合蛋白，其中COX-2为诱导型酶，研究发现COX-2与肿瘤的发生发展有关。Cyclooxygenase is a complete membrane combined protein. COX-2 is an induced enzyme that related with the progress and oncogenesis.

结论ILK和MMP-9蛋白过度表达可能分别在NSCLC的发生和发展过程中起重要作用。Conclusion The overexpression of ILK and MMP-9 genes may play an important role in the oncogenesis and development of NSCLC respectively.

比如说，如果你真正着迷于蛋白激酶生物化学，你不一定要在肿瘤学范围内研究激酶。If one is really, for instance, captivated by the biochemistry of protein kinases, you don't necessarily have to work on kinases in oncogenesis.

端粒酶激活可以维持增殖细胞染色体的稳定性，导致细胞永生化和肿瘤发生。Telomerase activation is essential for the stabilization of chromosomes in proliferation cells, leading to cellular immortality and oncogenesis.

结论ABCG2在肾癌组织中表达较高，其可能参与了肾癌的发生和发展，可以作为肾癌干细胞研究的重要标志物。ABCG2 may be closely related to the oncogenesis and progress of renal cancer, and may be an important factor in the research of renal cancer stem cell.

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综述细胞凋亡与肿瘤形成的关系，消化系统各种肿瘤的形成的相关因素和细胞凋亡中的作用。This paper is to summarize the relationship between cell apoptosis and oncogenesis and the action of varied digestive tumor related genes in apoptosis.

目前已发现这个病毒会改变细胞基因修补的功能，因而引起染色体的断裂及细胞基因的变化。We have found that HCV infection induces DNA breaks and enhances DNA mutation frequencies, thus explaining the mechanism of oncogenesis of this RNA virus.

与所有肿瘤一样，胰腺癌的发生也是一种多因素作用，多基因参与，多阶段形成的复杂病理过程。Like all the other cancers, the oncogenesis of pancreatic cancer is also a complicated pathological process involves many factors, many genes and many stages.

在食管鳞状细胞癌发生发展中，是否有错配修复缺陷途径的参与，是否存在基因组不稳定状态，MSI和LOH各自发挥了什么作用，都是需要阐明的问题。The role of MSI and LOH is not very clear in the oncogenesis and process of esophagus squamous cell carcinoma, as well as of MMR genes and genome instability.

在青蛙肾癌模型，从细胞核转移到卵母细胞瘤细胞据报道，扭转了瘤的发生，也导致了蝌蚪阶段的发展。In a frog renal carcinoma model, the transfer of nuclei from tumor cells into oocytes was reported to reverse oncogenesis and to direct development to the tadpole stage.

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目的观察空间诱变小鼠黑色素瘤B16细胞株的成瘤性，寻找免疫原性发生变异的细胞株。Objective To explore the oncogenesis of the melanoma B16 cell clones mutagenized by space environment, and find the B16 cell clones with remarkably mutated immunogenicity.

业已证明，HSP70在乳腺癌、肾癌、子宫癌、卵巢癌等肿瘤中表达增加，可能对肿瘤产生与发展具有意义。HSP70 has been found over-expressed in breast cancer, renal cancer, ovarian cancer, etc. and it has been suggested possibly participating in the oncogenesis of these cancers.

目前在分子水平上的研究显示肿瘤的发生与癌基因的激活和抑癌基因的失活以及细胞周期调节失控密切相关。It has been proved that the occurrence of oncogenesis is in close relationship with the oncogene activation, the tumor suppressor gene inactivation and the disorder of cell cycle modulation.