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在左边黑质处是正常数目的神经元，并且出现色素沉着。At the left, normal numbers of neurons substantia nigra are pigmented.

它是由于中脑塞梅林氏神经节中的神经细胞死亡导致的。It is caused by the death of nerve cells in a midbrain area called substantia nigra.

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而多种因素引起的脑内多巴胺能神经元变性、死亡则是PD的主要发病机制。The characteristic pathology in PD is the degeneration of dopamine neurons in the substantia nigra pars compacta.

本文报告了大白鼠脊髓胶状质内嵴突触的超微结构特征。Crest synapses of the substantia gelatinosa have been observed in rat spinal cord by means of electron microscopy.

结果黑质致密部中脑脑片可见典型的多巴胺神经元形态，小胶质细胞呈典型的静息状态。Results The typical dopaminergic neurons were observed at substantia nigra pars compacta in midbrain brain slices.

结论雌激素对雌性大鼠中脑黑质多巴胺能神经元具有保护作用。Conclusion Estrogen plays an important role in protecting the substantia nigral dopaminergic neuron of female rats.

她的研究对象是某个脑部区域的结构性病变，这部分在大脑深处，称为无名质。She looked for changes in structure in the brain area called the substantia innominata, an area deep in the brain.

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黑质的谷氨酸和GABA可能通过影响尾壳核DMT1-IRE和HP的表达影响纹状体尾壳核的铁代谢。Glutamate and GABA in the substantia nigra may affect iron metabolism through DMT1-IRE and HP in the caudate putamen nucleus.

结果与健康组比，PD人脑标本中缝背核、黑质以及纹状体的SERT标记信号呈不同程度降低。Results In comparison with healthy group, SERT decreased in dorsal raphe nucleus, substantia nigra and striatum in PD brains.

帕金森病是神经系统变性疾病，以黑质多巴胺能神经元变性缺失为特征。Parkinson's disease is resulted from the progressive degeneration and from the loss of dopaminergic neurons in Substantia Nigra.

结论黑质的谷氨酸和GABA可能通过影响尾壳核DMT1-IRE和HP的表达影响纹状体尾壳核的铁代谢。Conclusion Glutamate and GABA in the substantia nigra may affect iron metabolism through DMT1-IRE and HP in the caudate putamen nucleus.

解剖许多帕金森氏症患者的尸体后发现，黑质多巴胺神经元中有堆积成块的蛋白质。In many parkinson's cases, the damage can be seen in autopsies as clumps of proteins within the substantia nigra's dopaminergic neurons.

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结果帕金森病大鼠细胞凋亡数随着左旋多巴治疗的时间、剂量增加而增加。Results The number of substantia nigral apoptosis cells was increased and positively related to the changes of doses and durations of use of levodopa.

结论还原型谷胱甘肽可能对MPTP损伤的黑质多巴胺神经元凋亡有抗氧化保护作用。Conclusion Reduced glutathione may have protective effect on apoptosis and oxidative stress of substantia nigra neurons in MPTP induced PD mouse model.

观察模型大鼠黑质和纹状体多巴胺D1受体和多巴胺D2受体免疫组织化学染色强度。The immunohistochemical staining intensities of dopamine D1 receptor and dopamine D2 receptor in the striatum and substantia nigra of rat models were observed.

中脑的一些区域的多巴胺神经元，如腹侧被盖区和黑质致密部，在对奖赏期待中起关键作用。Dopamine neurons in parts of the midbrain, such as the ventral tegmental area and substantia nigra pars compacta, play a vital role in the expectation of reward.

外科医师将导管伸入脑左右两边的纹状体，那里是接受黑质神经元释出多巴胺的主要基底核脑区。Surgeons insert a catheter into the left and right striatum, the main recipients in the basal ganglia of the dopamine secreted by neurons of the substantia nigra.

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结论由MPTP制备的PD小鼠模型，导致中脑黑质多巴胺能神经元毁损的本质可能是细胞凋亡。Conclusion MPTP is a substance for developing animal model of PD, and apoptosis may be a main factor for MPTP's injuring dopaminergic neurons in the substantia nigra.

慢性缺血大鼠在阿朴吗啡的作用下，刻板动作行为的积分会明显高于正常对照组，提示黑质和纹状体的多巴胺系统功能受到了干扰。Ultrastructure changes in substantia nigra and striatum after chronic ischemia have an effects on behavior of stereotypy and change function of dopamine system in rats.

腹腔注射MPTP后，模型组鼠的黑质和蓝斑区的2-DG摄取量较对照组鼠明显增加，并表现出明显的行为异常反应。Following MPTP intoxication, 2-DG uptake was increased in the substantia nigra and locus ceruleus. At the same time, Obvious abnormal behavior of the animals was induced.