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低血氧症是较可靠及敏感的早期诊断指标。Hypoxemia is a reliable and sensitive index for its early diagnosis.
结果6例临床表现为咳嗽、气急、呼吸困难和低氧血征。Results These 6 cases all had cough, progressive dyspnea and hypoxemia.
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吸痰可导致低氧血症和组织缺氧。Arterial hypoxemia and tissue hypoxia maybe induced by tracheal suction.
兴奋呼吸和催醒期间须保持呼吸道通畅,提高吸入气氧浓度,避免低氧血症发生。To keep airway patent and clean, and increase oxygen concentration inhaled to avoid hypoxemia.
全麻后病人应加强呼吸监测及管理,防止低氧血症的发生。It is important to prevent hypoxemia after general anesthesia via intensive care for respiratory.
低氧血症在肝功能C级患者最为明显,肝性低氧血症与门脉高压相关。Hypoxemia occurred mainly in patients with Child Pugh C, which was correlated with portal hypertension.
高原肺水肿的主要症状是严重的肺动脉高压和重度的动脉低氧血症。The key features of HAPE are exaggerated pulmonary hypertension and arterial hypoxemia at high altitude.
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方法对28例急性一氧化碳中毒持续低氧血症患者的临床资料进行回顾分析。Methods The clinical date of 28 cases with continuous hypoxemia after ACMP were analyzed retrospectively.
病患在住院的第七天突发急性呼吸窘迫,并快速导致低血氧。Acute onset of respiratory distress occurred on his seventh hospital day and rapidly resulted in hypoxemia.
围手术期并发症包括低氧血症1例,胸腔积液2例,均经保守治疗后痊愈。Perioperative complication including hypoxemia in 1 case and pleural effusion in 2 cases were cured by conservative treatment.
睡眠低氧血症及其继发的病理生理改变引起的脑缺氧为发病的主要机制。Cerebral anoxia due to nocturnal hypoxemia and its consequential pathological changes were the main mechanisms of this desease.
除肺血管发育条件外,大量侧枝血管对肺血管内皮破坏,造成术后低氧血症。The damage of numeral collaterals to pulmonary vessels leads to pulmonary hypertension and hypoxemia during postoperative period.
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结论OSAHS患者心律失常的发生与呼吸暂停低通气指数,低氧血症及交感紧张密切相关。The occurrence of cardiac arrhythmia in patients with OSAHS is closely related with AHI and nocturnal hypoxemia and sympathetic tone.
目的探讨舒氧康静脉给氧对老年病人术后早期低氧血症的防治效果。Objective To observe the preventive effect of early postoperative hypoxemia in aged patients with oxygenator by intravenous oxygen delivery.
结论经面罩无创机械通气治疗急性心源性肺水肿起效快,能明显改善低氧血症,是抢救急性心源性肺水肿的重要手段之一。Conclusion NIPPV can improved hypoxemia more rapidly than conventional therapy and is one of the important means to rescue acute pulmonary edema.
目前研究认为低氧血症是刺激胎盘组织分泌激活素A增多的主要调节机制。Recent researches demonstrated that hypoxemia is a specific trigger for elevation of activin-A in fetal circulation and is a main regulatory mechanism for it.
故肝肺综合征实际上是基础肝脏疾病-肺血管扩张-动脉血氧合功能障碍构成的三联体。Therefore, the hepatopulmonary syndrome substantially is a triplet that consists of liver disease, dilatation of the pulmonary microcirculation, and hypoxemia.
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结论SARS低氧血症与肝转氨酶关系密切,在治疗低氧血症的同时,应注意保肝支持治疗。Conclusions Hypoxemia is closely associated with abnormal liver transaminase in SARS. When treating hypoxemia, attention should be paid to protecting the liver.
血流动力学改变、血氧不足、脑血管自动调节功能紊乱是其导致卒中的主要机制。The alteration of cerebral hemodynamics, hypoxemia , and dysfunction of cerebral autoregulation appear to be the main mechanisms of stroke in patients with SAS.
然而一名15岁的女孩除了产生呼吸道刺激的症状外,另有缺氧及主要影响右侧中、下肺之急性肺伤害。A 15-year-old girl however developed hypoxemia and acute lung injury primarily involved right middle and lower lungs in addition to symptoms of airway irritation.