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纤维朊原分子体积很大。The molecular volume of fibrinogen is large.

采用组织凝血活酶法测定血浆纤维蛋白原水平。Plasma fibrinogen levels were assayed by tissue thromboplastin.

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血尿酸和血浆纤维蛋白原二者间无相关性。There was no correlation between blood uric acid and fibrinogen.

其效应具有浓度依赖关系。The effects of bFGF and fibrinogen depend on their concentration.

腔隙性梗死患者的发病与血浆纤维蛋白原水平密切相关。The level of plasma fibrinogen is associated with lacunar infarction.

治疗组血纤维蛋白原水平降低显著，不良反应轻微。The serum fibrinogen level was decreased significantly in the treatment group.

入院后检测血浆纤维蛋白原水平。They were tested for plasma fibrinogen level right after admitted to hospital.

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在第二次实验中，纤维蛋白原的消耗被封闭了，所有动物均存活。This time, fibrinogen consumption was blocked, and all of the animals survived.

若纤维蛋白原减少者可输纤维蛋自原制剂。If the fibrinogen subtracter may lose the textile fiber egg from original preparation.

血浆纤维蛋白原在冠心病的发病中可能起重要作用。It is suggested that fibrinogen in plasma takes an important role in the the pathogenesis of CHD.

蝮蛇抗栓酶从蝮蛇蛇毒提取的具有降低纤维蛋白原和血液粘滞度的物质。Ahylysantinfarctase from snake venom extract with lower fibrinogen and blood viscosity of the material.

各组治疗后7天血浆纤维蛋白原水平较治疗前显著降低，治疗后14天血浆纤维蛋白原水平与治疗前相比无显著变化。The serum fibrinogen level was decreased significantly on 7th day both in A and B group but not on 14th day.

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结论发病后高纤维蛋白原水平为急性进展性脑梗死的危险因素。Conclusion The high levels of fibrinogen are one of the risk factors in acute progressive cerebral infarction.

目的探讨血浆纤维蛋白原水平与急性单纯闭合颅脑损伤患者严重程度的相关意义。Objective To explore correlation between the level of plasma fibrinogen and severity of craniocerebral injury.

采用比浊法测定兔血小板聚集率，双缩脲法测定血浆纤维蛋白原浓度。Platelet aggregation was measured turbimetrically and plasma fibrinogen level was determined by biuret method.

结论纤维蛋白原FGA基因复合杂合缺失是引起该家系先证者无纤维蛋白原血症的原因。Conclusion Inherited afibrinogenemia is caused by the compound heterozygous deletion in the fibrinogen FGA gene.

结果发现纤维蛋白原和蛋白C被消耗，各反映器官损伤的指标明显增高。The result was fibrinogen consumption, protein C consumption, and markers of organ damage all markedly increasing.

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重组巴曲酶在体外可使纤维蛋白凝固，在体内缩短小鼠出血时间。The purified recombinant protein converted fibrinogen into fibrin clot in vitro, and shortened bleeding time in vivo.

根据诱发疾病及DIC的发展阶段，合理使用肝素及补充纤维蛋白原、凝血因子是提高治愈率的重要手段。It was the important method of rising cure rate to use heparin rationally and supply fibrinogen and coagulation factors.

HID4则识别另一抗原决定簇，且与纤维蛋白原碎片X、Y、D等有交叉反应。HID4 recognizes another antigenic determinant and cross-reacts with fibrinogen degradation products X, Y and D fragments.