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比较梗塞侧与正常右侧的区别。Compare the infarcted side with the normal right side.

急性水肿性梗死组织可形成团块效应。The acutely edematous infarcted tissue may produce a mass effect.

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模型组和克罗卡林组脑切片上可见较大的苍白色梗死区。In the model and cromakalim groups, a large, pale, infarcted region was observed.

注意梗塞组织的肿胀和周围组织的变形。Notice the swelling of infarcted tissue and the distortion of surrounding tissue.

当组织梗塞或水肿时，就不能辨认出此区域内的沟回。As tissue has become infarcted and edematous, the sulcus is no longer identifiable.

图示暗红，灰白的肠梗死与底部淡粉红色的正常小肠对比。The dark red to grey infarcted bowel contrasts with the pale pink normal bowel at the bottom.

暗红甚至是灰色的梗死肠段与下部的粉红色正常肠段有着明显的区别。The dark red to grey infarcted bowel contrasts with the pale pink normal bowel at the bottom.

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说明预防脑梗塞后出血对改善患者预后具有重要意义。It is useful to prevent infarcted hemorrhage for the patients with brain infarction from his recovery.

结论康复功能训练可促进大鼠脑梗死的神经功能的恢复。Conclusion Rehabilitative training can promote the recovery of the cerebral infarcted neural functions in rats.

应用印压实验装置，对正常和陈旧性梗死离体心室肌试件进行蠕变试验。With the instrument of indentation, creep test was applied to the normal and old infarcted canine ventricular myocardium.

机械性能的改善可能由于植入细胞在梗死区造成的拱形结构所致。Improed passie mechanics may result in part from a mechanical buttressing of the infarcted wall by the transplanted cells.

目的观察同种异体心肌细胞移植对心肌梗死大鼠心肌的修复作用和心功能的影响。Objective To study the effects of transplanted allogenic cardiomyocytes on the repair and function of infarcted myocardium of rat.

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此外，缺乏MCD的大鼠，在其慢性心肌梗死的心衰进展过程中，功能显著降低。Furthermore, the development of heart failure in chronically infarcted mouse hearts is significantly decreased in mice lacking MCD.

对大脑进行的组织学切片染色结果显示梗塞灶位于尾状核及大脑半球外侧皮质。Cerebral tissue histologic studies show that the infarcted focus locates in caudate nucleus and lateral cortex of cerebral hemisphere.

正常、危险和梗死心肌血流量、微血管体密度差异显著。For the MBF and the myocardial micro vessel volume density, there was significant difference in normal, risk and infarcted myocardium.

结论自体骨髓间质干细胞移植能有效改善兔心肌梗死后左室收缩功能，减轻室腔扩大。Conclusions Autologous bone marrow stem stromal cells transplantation can improve systolic function and alleviate the enlargement of infarcted LV.

激素可阻断炎症反应过程，但同时也抑制了骨髓干细胞向梗死灶归巢。Hormone can stop the process of inflammation, but the ability of bone marrow stem cells homing to infarcted myocardium is inhibited at the same time.

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动物实验提示细胞移植可以修补坏死心肌，增加梗死区细胞数量，改善心功能。Animal experiments indicated that cell transplantation could repair necrotic myocardium, enhance the cells in infarcted zone and improve heart function.

增强后梗塞心肌信号率既高于增强前也明显高于其周围的正常心肌。After enhanced, the signal intensity ratio of the infarcted myocardium was significantly higher than both that before enhancement and that of the normal.

空白对照组、心肌梗死1周后处理组及心肌梗死2周后处理组，处理组经心外膜向心肌注射自体骨髓单核细胞。Autologous bone marrow MNCs were injected via the epicardium into the infarcted area in the latter two groups at 1 and 2 weeks after the infarction, respectively.