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H4组蛋白就是五个组蛋白团之一。Histone H4 is one of five histone groups.

本发明涉及组蛋白脱乙酰基酶的抑制。The invention relates to the inhibition of histone deacetylase.

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这是DNA分子与组蛋白相互作用的结果。It is the result of interaction between DNA and histone proteins.

组蛋白赖氨酸的甲基化在表观遗传调控中起着关键作用。Histone lysine methylation plays a key role in epigenetic regulation.

第一章是组蛋白分子伴侣的背景介绍。There are an introduction and overview of histone chaperone in chapter 1.

一个小小的分子称作一个乙酰基团，它黏在一个称作H4的特殊组蛋白上。A small molecule called an acetyl group attaches to a particular histone known as H4.

这一组蛋白的甲基化靶点就称为H3K9，其中K代表赖氨酸。In the histone code the methylation target is known as H3K9, where K stands for lysine.

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人端粒酶逆转录酶在宫颈癌组织中的表达及意义。Role of hTERT in apoptosis of cervical cancer induced by histone deacetylase inhibitor.

组蛋白赖氨酸去甲基化酶有可能成为一个新的抗肿瘤治疗靶标。Therefore, histone lysine demethylases may serve as a new target for anti-cancer therapy.

总结了组蛋白精氨酸甲基化修饰体系的最新研究进展。Here, the most recent progresses in the methylation studies of histone arginine were summa.

除了MHC外，6号染色体上的该区域也包含一组组蛋白基因。In addition to the MHC, this region on chromosome 6 also contains a cluster of histone genes.

为了逆转记忆遗失，医生在复合物中添加了组蛋白脱乙酰基酶抑制物。To reverse the memory loss, doctors inject a compound called a histone deacetylase inhibitor.

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组蛋白既保护DNA也在它们与DNA结合形成染色质时调节基因表达。Histone proteins both protect DNA and regulate genes as they combine with DNA to form chromatin.

SITR1是一种组蛋白脱乙酰基酶，一种能够阻断来自调节基因的转录因子的蛋白质。SITR1 is a histone deacetylase, a protein that blocks transcription factors from regulating genes.

本研究是对推测的小麦组蛋白乙酰转移酶及脱乙酰化酶基因功能研究的首次报道。This is a first report about function study of histone acetyltransferase and deacetylase in wheat.

组蛋白去乙酰化酶的抑制诱导组蛋白的高度乙酰化。The inhibition of the histone deacetylase enzymes induces hyperacetylation of the histone proteins.

更加专门化的组蛋白变体在一些谱系中进化到执行额外的功能。Further specializations of histone variants have evolved in some lineages to perform additional tasks.

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这里，我们报告了来自于人类CD4细胞中39个组蛋白修饰的分析的模式。Here, we report the patterns derived from the analysis of 39 histone modifications in human CD4 T cells.

第一部分工作是关于聚腺苷酸化对组蛋白H3.3的表达的调控。The first part is studying on"Regulation of the Expression of Histone H3.3 by Differential Polyadenylation".

组蛋白密码的这些改变可能改变基因表达模式以设定白血病发生状态。These alterations in the histone code may alter gene expression pattern to set the stage for leukemogenesis.