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方法分离、培养大鼠心脏微血管内皮细胞。Methods Mouse CMEC were isolated and cultured.
对微血管密度与VEGF、MMP9的关系进行分析。The relationship among MVD and VEGF, MMP9 was evaluated.
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病变的特征是微血管基底膜增厚而脆弱。The walls of the vessels become abnormally thick but weak.
各组微血管记数结果金雀黄素组最少。The count of microvessel was the fewest in genistein group.
我们检测了微血管浸润的作用。We investigated preoperative predictors of microvascular invasion.
有大量的增殖性巨大微血管横列在这些组织之间。A large number of proliferating macrocapillaries coursed among them.
微血管数是显著独立的预后因素。Microvessel count was a significantly independent prognostic factor.
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针刺后观察，其微血管病变得到改善。Afer acupuncture, the pathological changes of micrangium were improved.
根据临床及病理的表现，其诊断为持久性斑状微血管扩张症。According to the clinical and pathological findings, TMEP was diagnosed.
TMBG和TMP从心肌微血管水平对再灌注进行评价。TMBG and TMP provide the reperfusion evaluation on myocardial microvascular level.
微血管基底膜的增龄变化可能是其机制之一。The microvessel basement membrane changes with age might be the one of the mechanisms.
近年来研究表明，PEDF与糖尿病微血管病变关系密切。Recent studies showed that PEDF had a close relationship with diabetic microangiopathy.
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因此，该药可能能够改善扩心病患者的冠脉微血管功能。As a result, the drug appears to improve coronary microvascular function in these patients.
目的分离和培养人腹膜微血管内皮细胞。Objective To isolate and culture microvascular endothelial cells from human omental tissue.
所以显微血管性后骨间动脉皮瓣提供手部软组织重建一个可靠的选择。The free posterior interosseous flap provides a reliable option for coverage of hand wounds.
软组织对钝器伤的反应包括微血管反应和炎症性过程。The reaction in soft tissue healing process contains microvascular responses and inflammation.
结论糖尿病微血管病变是腔隙产生的危险因素之一。Conclusion Diabetic microvascular complication is one of the risk factors of lacunar infarction.
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赛庚啶可显著降低脑皮质及脑微血管中5-HT含量，并减轻脑继发性损害。Cyproheptadine reduced 5-HT levels in the brain cortex and BMVs, and the secondary brain damage.
检测皮肤表理层的黑色素，3D纹理、雀斑及微血管扩张……Checking the melanin in the skin inner underlayer, 3D texture, expansion of capillary vessel, etc.
结论生物散斑现象可作为研究微血管自律运动的新方法。Conclusion The phenomena of bio speckles can be used as a new method in the research on vasomotion.