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依普利酮是选择性的醛甾酮拮抗药。Eplerenone is a selective aldosterone antagonist.

在液体容量不足的患者,醛固酮水平应该降低。Aldosterone levels should be low in volume-replete patients.

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下一步,收集一定时间段的尿液测定醛固酮水平。Next, initiate timed urine collection to determine aldosterone levels.

目的豚鼠腹腔注射醛固酮建立膜迷路积水的动物模型。Objective To develop endolymphatic hydrops in both ears of guinea pigs by aldosterone.

阻断血管紧张素I向血管紧张素II的转化,防止肾上腺皮质分泌醛固酮。Block the conversion of ANG I to ANG II and prevent the secretion of aldosterone from the adrenal cortex.

在结合应激激素的同时,它还能够对其他激素产生响应,例如醛固酮和去氧皮质酮。As well as binding cortisol, it could respond to other hormones such as aldosterone and deoxycorticosterone.

选择性环氧合酶-2抑制剂对血压的影响是否可以用血浆醛固酮水平变化解释?。Can the blood pressure effects of COX-2 selective inhibitors be explained by changes in plasma aldosterone levels?

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如果经过容量替代治疗,患者尿液醛固酮水平仍高,那么就存在醛固酮的异常刺激。If urinary aldosterone levels are high despite volume replacement, there is an abnormal stimulation of aldosterone.

目的探讨肾素血管紧张素醛固酮系统在甲亢性心脏病发病中的作用。Objective To study the role of renin angiotensin aldosterone system in the pathogenesis of hyperthyroid heart disease.

肾外血容量减少是低血压、高醛固酮排泄、血钾丢失的一个可能原因。Extrarenal volume depletion is a possible reason for low blood pressure, high aldosterone excretion, and potassium loss.

IHA为主要类型,发病与异常的促醛固酮分泌因子的增加及基因变异有关。The etiology of IHA, the main subtype, is related with increased abnormal aldosterone excreted factor and genovariation.

目的探讨醛固酮对大鼠肾小球系膜细胞结缔组织生长因子基因表达的影响。To evaluate the effect of aldosterone on connective tissue growth factor mRNA expression in cultured rat mesangial cells.

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结论ET与肾功能、血压及紧素一血管紧张素一醛固酮系统关系密切。Conclusions The increase of plasma ET 1 may have an intimate relation to renal function and renin angiotensin aldosterone system.

结论①心力衰竭患者ACEI治疗后,不管是否加用螺内酯均可观察到醛固酮逃逸现象。Conclusion ①The aldosterone escape appears in congestive heart failure patients with ACEI treatment irrespective of spironolactone use.

卡托普利逆转压力负荷增加大鼠心肌纤维化的作用可能与降低血浆醛固酮水平有关。The effect of regressing myocardial fibrosis in the rat with CHF by captopril may be related to the decrease in plasma aldosterone level.

由于TALH或DCT不能保钠,因而会出现盐和水的丢失,继而会激活RAAS,使醛固酮水平升高。Salt and water depletion due to inability to conserve sodium in the TALH or DCT leads to activation of the RAAS and high aldosterone levels.

并且,四个月内肾激素和醛固酮指标的小幅度增长,也与服用利尿剂降血压所产生的效果差不多。And small increases in renin and aldosterone at four weeks are similar to that which occurs when diuretics are given to reduce blood pressure.

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结论安体舒通对肺纤维化有一定的保护作用,醛固酮可能参与肺纤维化的发病过程。Conclusion Spironolactone had some protective effects on pulmonary fibrosis, and aldosterone might play a role in the development of pulmonary fibrosis.

结论肾素血管紧张素醛固酮系统激活是甲亢性心脏病发病的重要原因之一。Conclusion Hyperthyroidism results in activation of renin angiotensin aldosterone system which contributes to the pathogenesis of hyperthyroid heart disease.

饮食的改变也使肾脏分泌出更多的肾激素,肾激素是一种蛋白质,它的激素醛固酮与高血压密切相关。The dietary change was also shown to cause the kidneys to release more of a protein called renin and its hormone aldosterone which is linked with high blood pressure.